Bee products to support the fight against cancer




Bilgi almak istediğiniz konuları buraya yazarak enter'e basınız

All types of cancer, the cancer spread and tumor growth, the use of bee products, half rates to slow down the disease resistance of the fracture treatment, a significant prolongation of life research on the acquisition and the time zones listed below.

Strengthens the immune system, plus support for aspects of cancer treatment is provided. Strength of the patient, the patient's quality of life and those listed görülür.Bu increase in the overall resistance, high morale and combat the disease can lead to powerful to have perseverance. Given the weight of the psychological aspect of the human from the bee products, plus the importance of these contributions is indisputable.

On the other hand, protecting the internal organs to the side effects of chemotherapy and radiotherapy, the patient, this page can be found in research on the effects of rapid recovery provider.

Bee products contain 200 times more antioxidant than vitamin E and flavonoids have value. Reduces the risk of cancer is also remarkable in this respect, in terms of the use of preventive medicine is concerned.
Important Note: Bee products, expected benefits may not occur in the same way for everyone. Before you use those products in general allergy to bee products are advised to consult an allergy specialist. The category of bee products in general is a treat in itself be considered as treatment support.

Some of them are on the subject of hundreds College Surveys:

Spread of cancer (metastasis) Cut in Half Slows:

Scientific publications of Oxford University, one of Japan Gifu Pharmaceutical Sciences, according to research reported in Oxfordjournals'da royal jelly shows anti-tumor effects. In addition to these effects on tumor growth slows down and stops the active ingredient of royal jelly, the HDA-10, and the replacement of the spread of cancer cells (metastasis) is also partially blocked. For more information:

Kuyushu University Prof. Nakaya and experimental studies of academic staff, royal jelly breast cancer cells with anti-dissemination activities has Cevreli-estrogens, which passes in front of it surrounds the effects are verifying. For more information:

The active ingredient of propolis Penetil caffeic acid ester (CAPE) on the effects on tumor and metastasis Prof. Memorial Hospital in Taiwan Hui-Fen Liao and cancer cells to the effects of oncologists propagation velocity of the content of the final report, propolis, rates of cancers up to 47-52 per cent decrease said. For more information:

Inhibit the growth of tumors:

Prof. M. Demestre, German, American and Italian universities, scientists have observed with the long-term effects of propolis extract. Try this as a result of long-term, propolis significantly suppressed tumor growth in 100 days of use, the type of cancer, according to this ratio shows small deviations from the average value is 47.5 per cent are explained.

For more information:

Brazilian Prof. Michelle C.Bufalo the results of the tests carried out with the participation of some scientists pioneered the scientific basis for a strong anti-tumor effects of propolis on paper emphasized. Anti-inflammatory, anti-microbial, anti-viral characteristics as noted. For more information:

Reduces Tumor robust and durable:

Study published in the December issue of Molecular Nutrition and Food Research, the active ingredient of propolis, and CAPE, the resulting use of propolis is a very interesting effect quertin'in are found. Accordingly, the active ingredients to build ways of tumors by blocking their blood, tumor development and durability of cutting way. For more information:

Survey carried out by an academic group in Croatia in July 2009 Another interesting effect of propolis are found. Extract Propolis is the protective effect of radiotherapy processes, implying a range of healthy tissue and cells, the result is taken. For more information:

Cancer Life Expectancy significantly upsetting:

Japan Fujisaki Institute, Hayashibara Biochemical studies Labaratuarları'nda tumor-bearing mice, in September 2003 on the use of royal jelly decreased DNA damage and prolonged survival of the mice said. However, according to this study, which is one of the major problems faced by cancer patients who were treated in the bone marrow as a result of the decline aktivasyonlarındaki reduction of red and white blood cells able to protect royal jelly. For more information: <- click here ..
Immunopharmacology International, April 2005 issue of the journal, published between 679-688 pages work against the formation of tumor growth and the effects of royal jelly is about. and Prof. Bincoletto an academic group's research and oxidative DNA degradation as a result, the mice were given royal jelly for 16 weeks significantly prolonged survival of reduction of the distortion of the DNA said. For more information:

Number of Lung Tumors by Kullanılmayanlara four times less than those used in Propolis ...

Department of Pharmacology, University of Oyama Japanese Science Man 10 According to research published in Science Direct, showing the effect of blocking the use of propolis in preventing the progression of lung cancer patients.

Tobacco carcinogens 7 weeks of the experiment based on the formation of tumors in the lungs of mice by exposing them to intense provided. Then, the rate of 100mg/kg per day for 16 weeks is given through the mouth of propolis. Number of tumors in mice exposed to carcinogen is not only releasing the propolis (mean 12.3), propolis is applied after carcinogen number of tumors in mice (mean, 3.5) is based on a surplus of around 4 times. In the conclusion, "According to the findings in cases of lung cancer in humans can be used as the content of propolis kemoprevensif" it said.

Click the link below for the full text of Araştıramnın:

Lung Tumor Growth on the instant success of the treatment applied Kanseri'nde Use of Propolis increases by 30 percent:
5 Bölümün'den Taiwanese Journal of Radiation Oncology, Radiation Research magazine's Man of Science (Japan) 45 according to research published in the number of pages 253-260, only measured after 48 hours of treatment with the tumor growth durdurulabilmesine 46 percent, but 60 percent from the same time period, the amount of propolis comes with. This rate rises to 67 percent after 72 hours. In contrast to tumor cells to radiation-induced damage to healthy cells the ratio is falling.

Drawing attention to the effects of the antioxidant content of propolis summarized in the conclusion of the study states:

"Lung cancer cells show a similar effect on the lethal effects of radiation. Radiotherapy in support of this point of view can be used together. Propolis, also can help in case of recurrence of the disease."

Click the link below to access the survey:

Kemoterapi'nin the liver and kidneys and Tissue Damage and Cell Death Induced Oxidative Print clearly Declining Use of Royal Jelly:
Erzurum Ataturk University, on 9 May 2011 a survey conducted by The Scientist under Oxidative Medicine and Cell Immunity received international publications. Ali Black, Nejdet Simsek, Emre Karakus, Sarah lightning, Adam Black, Ismail Can, Fikrullah short, the study conducted by Habib Turkeli Mehmet Emre and used in the treatment of cancer chemotherapy is an important content of the liver and kidneys against the destruction of Cisplatin'in recommend the use of royal jelly.

In the conclusion reads:

"As a result, cisplatin (CDDP) treatment, liver and kidneys at varying levels depending on the dose, while the use of royal jelly in subjects reduced the damage. Effect of reducing the protection of royal jelly, the death of healthy cells, antioxidants or free radicals that may be due to the effects of cleaning. Consequently , the use of royal jelly on the toxic effects of chemotherapy, liver and kidneys play a role in prevention. "

Click here to read the original English language were written to investigate ..

Note: The above research is diseased cells with blocking feature that emerged in this study the protective effect of healthy cells and its attendant benefits of royal jelly, the magnitude of the effects of selective spectacular clearly shows.

Chance of success with Royal Jelly Brain Tumor Tedavisi'nin Applicability and Growing:
Magazine's 28th issue of the Journal of the Neurological Sciences, pages 475-486, dated April 2011 a study published in the Kayseri Education and Research Hospital, Department of Radiation Oncology Specialist Physicians Jasmine Benderli Jahan, Erkan Göcek and the Department of Pathology, performed by Valerie Arsav.

According to the study, the use of royal jelly in the process of radiotherapy reduces radiation-induced damage to brain tissue. The amount of the dose administered to the tumor control are restricted to the damage taken increased by the use of royal jelly tumor control based on research results.

Some sections of the publication are as follows:

"Radiotherapy of primary central nervous system tumors and metastases is one of the treatment modalities. Radiotherapy aim to protect the brain by normal tissue as possible, to provide maximum dose of tumor tissue. Radiotherapy dose increased, a corresponding increase in local tumor control can be obtained. However, the normal tissue, resulting in the risk of complications increases. Consequently, tumor control, radiation tolerance of normal tissue in the brain depends on a sense. "

"Bee milk antioxidative effect, the structure of phenolic compounds or polyphenols possession available. Addition of these compounds, anti-inflammatory, anticarcinogenic, thermogenic, probiotic and antimicrobial properties that have the human, animal and in vitro studies have proven. Bee milk pharmacological activity of a large number of experimental animals have been identified , antitumoral, antimicrobial, inhibition of allergic reactions, blood cholesterol lowering effect of cell damage in patients with cancer and HIV prevention, wound healing and growth activator, vazodilatatif, such as anti-inflammatory activity has an effect. "

"Bee milk in experimental studies and toxic antineoplastic drugs with antioxidant effects against organ damage caused by ionizing radiation-induced brain injury is protective Despite the preventive role of the literature regarding whether or not could be detected. Bee milk is ionizing radiation-induced toxicity in patients treated with radiation to determine the preventive effect, it alternative natural antioxidants that work together to keep the light of the food is also important to take the place of supportive therapy. "

Proposed the use of royal jelly in the conclusion of the study, says: "According to the results of this research in the light of the findings of natural, non-royal jelly side effects in patients receiving radiotherapy to the brain in order to prevent the negative effects caused by radiation therapy before and after treatment can be recommended to give royal jelly. Study we have detected a positive impact, particularly antioxidative effect of royal jelly can be attributed to the beneficial biological activities. Topics related to the creation of more effective strategies will provide the design and implementation of clinical trials. "

Click here for the full text of the publication containing detailed explanation of the study:

google reklam 2-530

Arı sütüyle ilgili bazı araştırmalar/referanslar:

  • Risau W. Mechanisms of angiogenesis. Nature ( 1997;) 386:: 671–4. [CrossRef][Medline]
  • Yancopoulos GD, Davis S, Gale NW, Rudge JS, Wiegand SJ, Holash J. Vascular-specific growth factors and blood vessel formation. Nature ( 2000;) 407:: 242–8. [CrossRef][Medline]
  • Autiero M, Waltenberger J, Communi D, Kranz A, Moons L, Lambrechts D, et al. Role of PIGF in the intra- and inter-molecular cross talk between the VEGF receptors Flt1 and Flk1. Nat Med ( 2003;) 9:: 936–43. [CrossRef][Web of Science][Medline]
  • Folkman J, Klagsbrun M. Angiogenic factors. Science ( 1987;) 235:: 442–7. [Abstract/Free Full Text]
  • Ferrara N, Alitalo K. Clinical applications of angiogenic growth factors and their inhibitors. Nat Med ( 1999;) 5:: 1359–64. [CrossRef][Web of Science][Medline]
  • Kerbel RS. Tumor angiogenesis: past, present and the near future. Carcinogenesis ( 2000;) 21:: 505–15. [Abstract/Free Full Text]
  • Ferrara N. Vascular endothelial growth factor. Basic science and clinical progress. Endocr Rev ( 2004;) 25:: 581–611. [Abstract/Free Full Text]
  • Zachary I. VEGF signaling: integration and multi-tasking in endothelial cell biology. Biochem Soc Trans ( 2003;) 31:: 1171–7. [Web of Science][Medline]
  • Folkman J. Angiogenesis in cancer, vascular, rheumatoid and other disease. Nat Med ( 1995;) 1:: 27–31. [CrossRef][Web of Science][Medline]
  • Carmeliet P, Jain RK. Angiogenesis in cancer and other diseases. Nature ( 2000;) 407:: 249–57. [CrossRef][Medline]
  • Patel NG, Haydak MH, Gochnauer TA. Electrophoretic components of the proteins in honeybee larval food. Nature ( 1960;) 186:: 633–4. [CrossRef][Web of Science][Medline]
  • Shinoda M, Nakajin S, Oikawa T, Sato K, Kamogawa A, Akiyama Y. Biochemical studies on vasodilative factor in royal jelly [in Japanese]. Yakugaku Zasshi ( 1978;) 98:: 139–45. [Web of Science][Medline]
  • Kawamura J. Influence of gelee royale on embryos [in Japanese]. J Showa Med Assoc ( 1961;) 20:: 1465–71.
  • Yatsunami K. Echigo T. Antibacterial action of royal jelly. Bull Fac Agr Tamagawa Univ ( 1985;) 25:: 13–22.
  • Tamura T, Fujii A, Kuboyama N. Antitumor effect of royal jelly [in Japanese]. Folia Pharmacol Japon ( 1987;) 89:: 73–80. [CrossRef]
  • Townsend GF, Morgan JF, Hazlett B. Activity of 10-hydroxydecenoic acid from royal jelly against experimental leukemia and ascetic tumors. Nature ( 1959;) 183:: 1270–1. [CrossRef][Medline]
  • Townsend GF, Morgan JF, Tolnai S, Hazlett B, Morton HJ, Shuel RW. Studies on the in vitro antitumor activity of fatty acids I. 10-hydroxy-2-decenoic acid from royal jelly. Cancer Res ( 1960;) 20:: 503–10. [Abstract/Free Full Text]
  • Nakajin S, Okiyama K, Yamashita S, Akiyama Y, Shinoda M. Effect of royal jelly on experimental hypercholesterolemia in rabbits [in Japanese]. Yakugaku Zasshi ( 1982;) 36:: 65–9.
  • Fujii A, Kobayashi S, Kuboyama N, Furukawa Y, Kaneko Y, Ishihama S, et al. Augmentation of wound healing by royal jelly (RJ) in streptozotocin-diabetic rats. Jp J Pharmacol ( 1990;) 53:: 331–7. [CrossRef]
  • Takenaka T. Chemical composition of royal jelly. Honeybee Sci ( 1982;) 3:: 69–74.
  • Echigo T, Takenaka T, Yatsunami K. Comparative studies on chemical composition of honey, royal jelly and pollen loads. Bull Fac Agr Tamagawa Univ ( 1986;) 26:: 1–12.
  • Townsend GF, Brown WH, Felauer EE, Hazlett B. Studies on the in vitro antitumor activity of fatty acids. The esters of acids closely related to 10-hydroxy-2-decenoic acid from royal jelly against transplantable mouse leukemia. Can J Biochem Physiol ( 1961;) 39:: 1765–70. [Medline]
  • Maeda T, Kuroda H, Motoyoshi K. Effects of royal jelly and 10-hydroxy decenoic acid on the sebaceous glands of hamster ear. Jpn J Dermatol ( 1987;) 98:: 469–75.
  • Koya-Miyata S, Okamoto I, Ushio S, Iwaki K, Ikeda M, Kuramoto M. Identification of a collagen production-promoting factor from an extract of royal jelly and its possible mechanism. Biosci Biotechnol Biochem ( 2004;) 68:: 767–73. [CrossRef][Medline]
  • Blum MS, Novak AF, Taber S III. 10-Hydroxy-2-decenoic acid, an antibiotic found in royal jelly. Science ( 1959;) 130:: 452–3. [Abstract/Free Full Text]
  • Daniela P, Silvia P, Valentina U, GianLuigi C, Paola M. Pro-collagen I COOH-terminal trimer induces directional migration and metalloproteinases in breast cancer cells. J Biochem ( 2003;) 278:: 3639–47.
  • Martin MU, Ruth M, Ingrid F, Tatyana S, Jaroslav C, Roman AB, et al. Valproic acid inhibits angiogenesis in vitro and in vivo. J Nutr ( 2004;) 134:: 299–307. [Abstract/Free Full Text]
  • Samid D, Shack S, Sherman LT. Phenylacetate: a novel nontoxic inducer of tumor cell differentiation. Cancer Res ( 1992;) 52:: 1988–92. [Abstract/Free Full Text]
  • Patricia AM, Danilo Z, Clemet I, Mary MV, Suzanne FS, Sibel OM, et al. Isomers of conjugated linoleic acid differ in their effects on angiogenesis and survival of mouse mammary adipose vasculature. J Nutr ( 2004;) 134:: 299–307. [Abstract/Free Full Text]
  • Wright TJ, Leach L, Shaw PE, Jones P. Dynamics of vascular endothelial-cadherin and beta-catenin localization by vascular endothelial growth factor-induced angiogenesis in human umbilical vein cells. Exp Cell Res ( 2002;) 280:: 159–68. [CrossRef][Web of Science][Medline]
  • Lin MT, Yen ML, Lin CY, Kuo ML. Inhibition of vascular endothelial growth factor-induced angiogenesis by resveratrol through interruption of Src-dependent vascular endothelial cadherin tyrosine phosphorylation. Mol Pharmacol ( 2003;) 64:: 1029–36. [Abstract/Free Full Text]
  • Beecken WD, Engl T, Hofmann J, Jonas D, Blaheta R. Clinical relevance of serum angiogenic activity in patients with transitional cell carcinoma of the bladder. J Cell Mol Med ( 2005;) 9:: 655–61. [CrossRef][Web of Science][Medline]
  • Oak JH, Nakagawa K, Oikawa S, Miyazawa T. Amadori-glycated phosphatidylethanolamine induces angiogenic differentiations in cultured human umbilical vein endothelial cells. FEBS Lett ( 2003;) 555:: 419–23. [CrossRef][Web of Science][Medline]
  • Meghna UN, Ulhas PN. Junctional adhesion molecule-A-induced endothelial cell migration on vitronectin is integrin {alpha}β specific. J Cell Sci ( 2005;) 119:: 490–9. [CrossRef][Web of Science]
  • Tokunaga KH, Yoshida C, Suzuki KM, Maruyama H, Futamura Y, Araki Y, et al. Antihypertensive effect of peptides from royal jelly in spontaneously hypertensive rats. Biol Pharm Bull ( 2004;) 27:: 189–92. [CrossRef][Web of Science][Medline]
  • Tamarat R, Silvestre JS, Durie M, Levy BI. Angiotensin II angiogenic effect in vivo involves vascular endothelial growth factor- and inflammation-related pathways. Lab Invest ( 2002;) 82:: 747–56. [Web of Science][Medline]
  • Nadal JA, Scicli GM, Carbini LA, Scicli AG. Angiotensin II stimulates migration of retinal microvascular pericytes: involvement of TGF-beta and PDGF-BB. Am J Physiol Heart Circ Physiol ( 2002;) 282:: 739–48.

Propolis ile ilgili bazı araştırma/referanslar:

  • Bankova VS, Castro SL, Marcucci MC. Propolis: recent advances in chemistry and plant origin. Apidologie ( 2000;) 31:: 3–15. [CrossRef]
  • Banskota AH, Tezuka Y, Kadota S. Recent progress in pharmacological research of propolis. Phytother Res ( 2001;) 15:: 561–71. [CrossRef][Web of Science][Medline]
  • Huang MT, Ma W, Yen P, Xie JG, Han J, Frenkel K, et al. Inhibitory effects of caffeic acid phenethyl ester (CAPE) on 12-O-tetradecanoylphorbol-13-acetate-induced tumor promotion in mouse skin and the synthesis of DNA, RNA and protein in HeLa cells. Carcinogenesis ( 1996;) 17:: 761–5. [Abstract/Free Full Text]
  • Sforcin JM, Kaneno R, Funari SRC. Absence of seasonal effect on the immunomodulatory action of Brazilian propolis on natural killer activity. J Venom Anim Toxins ( 2002;) 8:: 19–29.
  • Scheller S, Gazda G, Pietsz G, Gabrys J, Szumlas J, Eckert L, et al. The ability of ethanol extract of propolis to stimulate plaque formation in immunized mouse spleen cells. Pharmacol Res Commun ( 1988;) 20:: 323–8. [Web of Science][Medline]
  • Bazo AP, Rodrigues MAM, Sforcin JM, Camargo JLV, Ribeiro LR, Salvadori DMF. Protective action of propolis on the rat colon carcinogenesis. Teratog Carcinog Mutagen ( 2002;) 22:: 183–94. [CrossRef][Web of Science][Medline]
  • Alves De Lima RO, Bazo AP, Said RA, Sforcin JM, Bankova V, Darros BR, et al. Modifying effect of propolis on dimethylhydrazine-induced DNA damage but not colonic aberrant crypt foci in rats. Environ Mol Mutagen ( 2005;) 45:: 8–16. [CrossRef][Web of Science][Medline]
  • Bassani-Silva S, Sforcin JM, Amaral AS, Gaspar LFJ, Rocha NS. Propolis effect in vitro on venereal transmissible canine tumor. Rev Port Ciênc Vet . in press.
  • Lima ME, Rissino JD, Guimarães AC, Overal DJ, Khayat AS, Souza PC, et al. Drifter technique: a new method to obtain metaphases in HEp-2 cell line cultures. Braz Arch Biol Technol ( 2005;) 48:: 537–40.
  • Orsi RO, Sforcin JM, Funari SRC, Bankova V. Effects of Brazilian and Bulgarian propolis on bactericidal activity of macrophages against Salmonella typhimurium. Int Immunopharmacol ( 2005;) 5:: 359–68. [CrossRef][Web of Science][Medline]
  • Boudourova-Krasteva G, Bankova V, Sforcin JM, Nikolova N, Popov S. Phenolics from Brazilian propolis. Z Naturforsch ( 1997;) 52c:: 676–9.
  • Bankova V, Boudourova–Krasteva G, Popov S, Sforcin JM, Funari SRC. Seasonal variations in essential oil from Brazilian propolis. J Essent Oil Res ( 1998a;) 10:: 693–6.
  • Bankova V, Boudourova–Krasteva G, Popov S, Sforcin JM, Funari SRC. Seasonal variations of the chemical composition of Brazilian propolis. Apidologie ( 1998b;) 29:: 361–7. [CrossRef]
  • ZarR JH. Biostatistical Analysis ( 1996;) 2nd. Englewood Cliffs: Prentice-Hall. 718.
  • Bankova V, Boudourova-Krasteva G, Sforcin JM, Frete X, Kujumgiev A, Maimoni-Rodella R, et al. Phytochemical evidence for the plant origin of Brazilian propolis from São Paulo state. Z Naturforsch ( 1999;) 54c:: 401–5.
  • Bankova V. Recent trends and important developments in propolis research. Evid Based Complement Alternat Med ( 2005;) 2:: 29–32. [Abstract/Free Full Text]
  • Jin UH, Chung TW, Kang SK, Suh SJ, Kim JK, Chung KH, et al. Caffeic acid phenyl ester in propolis is a strong inhibitor of matrix metalloproteinase-9 and invasion inhibitor: isolation and identification. Clin Chim Acta ( 2005;) 362:: 57–64. [CrossRef][Web of Science][Medline]
  • Lee YJ, Kuo HC, Chu CY, Wang CJ, Lin WC, Tseng TH. Involvement of tumor suppressor protein p53 and p38 MAPK in caffeic acid phenethyl ester-induced apoptosis of C6 glioma cells. Biochem Pharmacol ( 2003;) 66:: 2281–9. [CrossRef][Web of Science][Medline]
  • Aso K, Kanno S, Tadano T, Satoh S, Ishikawa M. Inhibitory effect of propolis of the growth of human leukemia U937. Biol Pharm Bull ( 2004;) 27:: 727–30. [CrossRef][Web of Science][Medline]
  • Kuo HC, Kuo WH, Lee YJ, Lin WL, Chou FP, Tseng TH. Inhibitory effect of caffeic acid phenethyl ester on the growth of C6 glioma cells in vitro and in vivo. Cancer Lett ( 2006;) 234:: 199–208. [CrossRef][Web of Science][Medline]
  • Lee YT, Don MJ, Hung PS, Shen YC, Lo YS, Chang KW, et al. Cytotoxicity of phenolic acid phenethyl esters on oral cancer cells. Cancer Lett ( 2005;) 223:: 19–25. [CrossRef][Web of Science][Medline]
  • Liao HF, Chen YY, Liu JJ, Hsu ML, Shieh HJ, Liao HJ, et al. Inhibitory effect of caffeic acid phenethyl ester on angiogenesis, tumor invasion, and metastasis. J Agr Food Chem ( 2003;) 51:: 7907–12. [CrossRef]
  • Orsolic N, Saranovic AB, Basic I. Direct and indirect mechanism(s) of antitumour activity of propolis and its polyphenolic compounds. Planta Med ( 2006;) 72:: 20–7. [CrossRef][Web of Science][Medline]
  • Shimizu K, Das SK, Hashimoto T, Sowa Y, Yoshida T, Sakai T, et al. Artepillin C in Brazilian propolis induces G(0)/G(1) arrest via stimulation of Cip1/p21 expression in human colon cancer cells. Mol Carcinog ( 2005;) 44:: 293–9. [CrossRef][Web of Science][Medline]
  • Kimoto T, Aga M, Hino K, Koya-Miyata S, Yamamoto Y, Micallef MJ, et al. Apoptosis of human leukemia cells induced by Artepillin C, an active ingredient of Brazilian propolis. Anticancer Res ( 2001;) 21:: 221–8. [Web of Science][Medline]
  • Inokuchi Y, Shimazawa M, Nakajima Y, Suemori S, Mishima S, Hara H. Brazilian green propolis protects against retinal damage in vitro and in vivo. Evid Based Complement Alternat Med ( 2006;) 3:: 71–7. [Abstract/Free Full Text]
  • Trusheva B, Popova M, Bankova V, Simova S, Marcucci MC, Miorin PL, et al. Bioactive constituents of Brazilian red propolis. Evid Based Complement Alternat Med ( 2006;) 3:: 249–54. [Abstract/Free Full Text]
  • Missima F, Sforcin JM. Green Brazilian propolis action on macrophages and lymphoid organs of chronically stressed mice. Evid Based Complement Alternat Med . in press.
  • Sforcin JM. Propolis and the immune system: a review. J Ethnopharmacol ( 2007;) 113:: 1–14. [CrossRef][Web of Science][Medline]